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Empagliflozin's Novel Cardioprotective Mechanism in Non-Diabetic Myocardial Infarction with Acute Hyperglycemyocardial Infarction

Abstract

Arundhati Guru

Individuals who experienced both acute myocardial infarction and hyperglycaemic myocardial infarction upon admission to the hospital had a worse prognosis than those who did not. The ability of sodium-glucose co-transporter-2 myocardial infarction inhibitors to treat acute hyperglycaemic myocardial infarction and the underlying processes remains unknown. When non-diabetic myocardial infarction caused by a glucose injection cooccurred with acute hyperglycaemic myocardial infarction in WT myocardial infarction. The survival rate was lower than in the control group. After using empagliflozin, a notable improvement in LV function and survival was seen. Also, when a myocardial infarction was coupled with an acute hyperglycaemic myocardial infarction, empagliflozin decreased fibrosis and autophagy of border cardiac tissue.

अस्वीकृति: इस सारांश का अनुवाद कृत्रिम बुद्धिमत्ता उपकरणों का उपयोग करके किया गया है और इसे अभी तक समीक्षा या सत्यापित नहीं किया गया है।

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